Update on Myocarditis

Link to 45-minute lecture by pediatric cardiologist Kirk Milhoan, MD, PhD, FAAP, FACC

Exactly three months ago, I posted an interview with my husband, pediatric cardiologist Kirk Milhoan, MD, PhD, FAAP, FACC on myocarditis related to covid-19 infection and “vaccination.” I’m now pleased to post a lecture he just delivered on this subject. It’s worthy of 45 minutes of your time, as this subject can be confusing and he attempts to address and be fair to numerous relevant studies in the medical literature. In the interest of time, my synopsis of his lecture follows.

I’ll repeat the same author’s note from three months ago: Doctors and scientists are struggling with terminology related to the covid-19 “vaccinations.” Using the term “vaccine” implies the traditional vaccine mechanism of action, where an antigen is introduced to which the body produces a desired antibody response. Four of the currently available covid-19 “vaccines” (Pfizer, Moderna, Johnson and Johnson, and Astra-Zeneca) involve the injection of genetic material which results in the cells of the body producing a protein, specifically the spike protein, to which the body then produces antibodies. We believe that use of the traditional terminology causes misunderstanding. Given their entirely different mechanism of action and thus different risk profile from traditional vaccines, the covid-19 “vaccination” products will be referred to with quotation marks in this essay.

Lecture synopsis:

·      The spike protein is cardiotoxic (it interacts with and activates toll-like receptor 4, TLR4, inducing inflammation via IL1-B. This is similar to the reaction to lipopolysaccharide, which is associated with severe systemic gram-negative bacterial infections). Therefore, cardiac damage can be seen both after infection and after “vaccination” with genetic products which result in the body’s cells producing spike protein.

·      Data supports a dose-response relationship to this cardiotoxin. Infection likely results in the smallest dose exposure to spike protein, as vaccination results in spike protein production for an as yet unknown period of time. Repeated vaccinations result in a greater incidence of damage. Vaccination with Moderna, which has about three times the mRNA than Pfizer, correlates with greater observed damage.

·      Vulnerability to cardiac damage is related to both age and gender, with young males (under 40) having the greatest risk.

·      Damage can be subclinical, meaning not observable by symptoms and many common tests. In a study of elite athletes after covid-19 infection, approximately 50% of cases of myocarditis were missed with screening for symptoms or tests such as EKG, troponin level, or echocardiogram. Cardiac MRI was required to detect all cases.

·      Late-gadolinium enhancement on cardiac MRI correlates with sudden cardiac death.

·      An autopsy of an individual that died after vaccination demonstrated atrial myocarditis. Damage to the sinus node, or pacemaker of the heart, is a conceivable mechanism for abnormal heart rhythms, even resulting in sudden cardiac death.

·      All-cause mortality in children in the EU has significantly increased and reports of myo- and peri-carditis to the Vaccine Adverse Events Reporting System (VAERS) have exponentially increased subsequent to the introduction of the “vaccine” products. This is cause for tremendous concern.

·      Proposing “vaccination,” which has a risk of myocarditis, in order to somehow prevent myocarditis violates the principle of “first, do no harm.” Exposing someone to a known risk in order to prevent an unknown risk is a violation of medical ethics. Most children now have natural immunity to SARS-CoV-2 and the current “vaccines” have not been demonstrated to prevent infection.

·      Cardiac MRI is required for initial diagnosis of all cases of infection or “vaccine”-induced myocarditis. (Incidence is approximately 1:50 for infection and for first-dose Pfizer. Incidence increases with Moderna and subsequent doses of either vaccine). Cardiac MRI is also required for long-term surveillance of those initially diagnosed with myocarditis.